Rose Ghemrawi
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Endoplasmic reticulum stress in metabolic disorders
R Ghemrawi, SF Battaglia-Hsu, C Arnold
Cells 7 (6), 63, 2018
Decreased vitamin B12 availability induces ER stress through impaired SIRT1-deacetylation of HSF1
R Ghemrawi, S Pooya, S Lorentz, G Gauchotte, C Arnold, JL Gueant, ...
Cell death & disease 4 (3), e553-e553, 2013
Methionine synthase and methionine synthase reductase interact with MMACHC and with MMADHC
C Bassila, R Ghemrawi, J Flayac, DS Froese, MR Baumgartner, ...
Biochimica et Biophysica Acta (BBA)-Molecular Basis of Disease 1863 (1), 103-112, 2017
Inherited disorders of cobalamin metabolism disrupt nucleocytoplasmic transport of mRNA through impaired methylation/phosphorylation of ELAVL1/HuR
SF Battaglia-Hsu, R Ghemrawi, D Coelho, N Dreumont, P Mosca, ...
Nucleic acids research 46 (15), 7844-7857, 2018
SIRT1 activation rescues the mislocalization of RNA-binding proteins and cognitive defects induced by inherited cobalamin disorders
R Ghemrawi, C Arnold, SF Battaglia-Hsu, G Pourié, I Trinh, C Bassila, ...
Metabolism 101, 153992, 2019
Endoplasmic reticulum stress in metabolic disorders. Cells 7 E63
R Ghemrawi, SF Battaglia-Hsu, C Arnold
Methyl donor deficiency impairs bone development via peroxisome proliferator‐activated receptor‐γ coactivator‐1α–dependent vitamin D receptor pathway
E Feigerlova, L Demarquet, H Melhem, R Ghemrawi, SF Battaglia‐Hsu, ...
The FASEB Journal 30 (10), 3598-3612, 2016
Endoplasmic Reticulum Stress and Unfolded Protein Response in Neurodegenerative Diseases
R Ghemrawi, M Khair
Int J Mol Sci 21 (17), E6127, 2020
MiR-16, but Not MiR-519, Suppresses Tumor Cell Proliferation in Meningiomas via HuR Inhibition
J Casse, A Oussalah, C Vigouroux, L Brochin, D Helle, R Ghemrawi, ...
Methyl donor deficiency impairs differentiation of pre-osteoblasts through disruption of functional interaction between peroxisome proliferator-activated receptor-gamma …
E Feigerlova, R Ghemrawi, H Melhelm, S Battaglia-Hsu, L Demarquet, ...
54th Annual ESPE 84, 2015
La carence en vitamine B12 induit un stress du réticulum endoplasmique dû à une diminution de la déacétylase SIRT1 et une augmentation de l'acétylation de HSF1
RI Ghemrawi
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